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bacteria:t3e:xops [2020/08/29 23:47] sujan.timilsina |
bacteria:t3e:xops [2022/07/19 12:56] rkoebnik [Biological function] |
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Author: [[https:// | Author: [[https:// | ||
Internal reviewer: [[https:// | Internal reviewer: [[https:// | ||
- | Expert reviewer: Sujan Timilsina | + | Expert reviewer: |
Class: XopS\\ | Class: XopS\\ | ||
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=== How discovered? === | === How discovered? === | ||
- | Although no homology was found between XopS and other T3E effectors, //xopS //was putatively identified by the presence of a plant-inducer | + | Although no homology was found between XopS and other T3E effectors, //xopS //was putatively identified by the presence of a plant-induced |
=== (Experimental) evidence for being a T3E === | === (Experimental) evidence for being a T3E === | ||
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To study the contribution of the T3Es to bacterial virulence, the effector gene was individually deleted in //Xcv// strain 85‐10, and the mutant was inoculated into leaves of susceptible ECW pepper plants. In addition, induction of the HR in pepper ECW‐10R was analyzed, which is based on the recognition of the T3E AvrBs1 by the Bs1 resistance gene (Schulze //et al//., 2012). Schulze et al. 2012 studied XopS along with XopB in their study. Deletion of //xopB// or //xopS// led to significantly reduced disease symptoms, whereas the HR induction was not impaired. The mutant phenotypes of 85-10Δ// | To study the contribution of the T3Es to bacterial virulence, the effector gene was individually deleted in //Xcv// strain 85‐10, and the mutant was inoculated into leaves of susceptible ECW pepper plants. In addition, induction of the HR in pepper ECW‐10R was analyzed, which is based on the recognition of the T3E AvrBs1 by the Bs1 resistance gene (Schulze //et al//., 2012). Schulze et al. 2012 studied XopS along with XopB in their study. Deletion of //xopB// or //xopS// led to significantly reduced disease symptoms, whereas the HR induction was not impaired. The mutant phenotypes of 85-10Δ// | ||
+ | |||
+ | XopS< | ||
=== Localization === | === Localization === | ||
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=== Interaction partners === | === Interaction partners === | ||
- | Unknown. | + | XopS< |
===== Conservation ===== | ===== Conservation ===== | ||
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=== In xanthomonads === | === In xanthomonads === | ||
- | Yes (//e.g.//, // | + | Yes (//e.g.//, // |
=== In other plant pathogens/ | === In other plant pathogens/ | ||
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Barak JD, Vancheva T, Lefeuvre P, Jones JB, Timilsina S, Minsavage GV, Vallad GE, Koebnik R (2016). Whole-genome sequences of // | Barak JD, Vancheva T, Lefeuvre P, Jones JB, Timilsina S, Minsavage GV, Vallad GE, Koebnik R (2016). Whole-genome sequences of // | ||
- | Fonseca NP, Patané JSL, Varani AM, Felestrino EB, Caneschi WL, Sanchez AB, Cordeiro IF, Lemes CGC, Assis RAB, Garcia CCM, Belasque Jr. J, Martins Jr J, Facincani AP, Ferreira RM, Jaciani FJ, Almeida NF, Ferro JA, Moreira LM, Setubal JC. (2019). Analyses of seven new genomes of // | + | Fonseca NP, Patané JSL, Varani AM, Felestrino EB, Caneschi WL, Sanchez AB, Cordeiro IF, Lemes CGC, Assis RAB, Garcia CCM, Belasque Jr. J, Martins Jr J, Facincani AP, Ferreira RM, Jaciani FJ, Almeida NF, Ferro JA, Moreira LM, Setubal JC (2019). Analyses of seven new genomes of // |
+ | |||
+ | Raffeiner M, Üstün S, Guerra T, Spinti D, Fitzner M, Sonnewald S, Baldermann S, Börnke F (2022). The // | ||
Schulze S, Kay S, Büttner D, Egler M, Eschen-Lippold L, Hause G, Krüger A, Lee J, Müller O, Scheel D, Szczesny R, Thieme F, Bonas U (2012). Analysis of new type III effectors from // | Schulze S, Kay S, Büttner D, Egler M, Eschen-Lippold L, Hause G, Krüger A, Lee J, Müller O, Scheel D, Szczesny R, Thieme F, Bonas U (2012). Analysis of new type III effectors from // | ||